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NUMERICAL ANALYSIS IN ARTERIAL STENOSIS AFFECTED BY ISCHEMIC HEART DISEASE USING FINITE VOLUME METHOD

BAREKENG: Jurnal Ilmu Matematika dan Terapan · Vol. 18 No. 1 (2024) · pp. 0179–0192

DOI
10.30598/barekengvol18iss1pp0179-0192
ISSN
2615-3017, 1978-7227
Published
2024-03-01
Publisher
PATTIMURA UNIVERSITY
Language
eng
License
creativecommons.org/licenses/by-sa/4.0

Abstract

Atherosclerosis is the narrowing of blood vessels caused by the buildup of cholesterol plaque on the walls of the arteries. Excessive buildup of cholesterol plaque disrupts the circulatory system, thereby affecting the speed and pressure of blood flow. In the long term, atherosclerosis can cause ischemic heart disease. This study aims to analyze the influence of stenosis, initial velocity, and diameter on the velocity and pressure of blood flow in narrowed arteries that cause ischemic heart disease. In this research, a Navier Stoke mathematical equation model was built which was solved using the finite volume method with SIMPLE discretization (Semi Implicit Method for Pressure Linked Equations). Finite volume methods are used to analyze unstructured objects such as blood flow. SIMPLE discretization is implemented simply in two and three dimensions with a system of equations containing fluid motion. Matlab and Fluent are software used for process simulations, Matlab for visualizing graphs of numerical calculation results, and Fluent for visualizing blood flow. Based on the simulation results, it can be concluded that the smaller the diameter, the greater the stenosis, and the greater the initial velocity, the greater the blood flow velocity. On the contrary, the diameter gets smaller, the stenosis gets bigger, and the initial velocity gets bigger so that the pressure on the blood flow gets smaller. Blood flow simulation has the potential to contain ischemic heart disease if the maximum speed produced is greater than the maximum normal blood speed, namely 0.45 m/s.

Collaboration network of the authors

40 authors · 133 co-authorship links · 5 across different organisations · showing the 40 most connected

Hover or tap a name to highlight its co-authors. Double-click to open the author's page.

  • Universitas Halu Oleo
  • Universitas Jember
  • Institut Agama Islam Negeri Kendari
  • Affiliation unknown
  • Different organisations
  • Same organisation
  • Affiliation unknown
  • Thicker line = more joint articles · bigger circle = more articles
Show as table
Co-authorOrganisationJoint articles
Edy WihardjoAffiliation unknown5
Dafik DafikUniversitas Jember4
Rafiantika Megahnia PrihandiniAffiliation unknown4
Saddam HussenAffiliation unknown4
Andi NurmasUniversitas Halu Oleo3
Muhammad Arief DirgantoroUniversitas Halu Oleo3
La Ode SafuanUniversitas Halu Oleo2
Waode Siti Anima HiseinUniversitas Halu Oleo2
AGUNG YUSWANAUniversitas Halu Oleo2
TERRY PAKKIUniversitas Halu Oleo2
Namriah NamriahUniversitas Halu Oleo2
Muslihatul AimaAffiliation unknown2
IMRAN SUBAIRUniversitas Halu Oleo1
SULWAN SULWANAffiliation unknown1
TRESJIA CORINA RAKIANAffiliation unknown1
Idrus SalamUniversitas Halu Oleo1
Agustono SlametUniversitas Halu Oleo1
MUSLIMAH MUSLIMAHAffiliation unknown1
FATKUR RAHMANUniversitas Halu Oleo1
I GUSTI RAY SADIMANTARAUniversitas Halu Oleo1
Sitti NurhalimahInstitut Agama Islam Negeri Kendari1
Tresjia Corina RakianUniversitas Halu Oleo1
Sitti LeomoUniversitas Halu Oleo1
Laode Muh. Harjoni KWAffiliation unknown1
Musadia AfaAffiliation unknown1
Nini Mila RahniUniversitas Halu Oleo1
LA KARIMUNAUniversitas Halu Oleo1
REZKI MUSAHIDINAffiliation unknown1
D DafikAffiliation unknown1
Putri Nur AmalinaAffiliation unknown1
Deddy SetyawanAffiliation unknown1
Anis Nur AfniAffiliation unknown1
Ermita Rizki AlbirriAffiliation unknown1
Girlyas Rasta YuntaAffiliation unknown1
Nira Nityasa SarantaAffiliation unknown1

Affiliations come from OpenAlex; journal OAI-PMH metadata carries none. Authors without an OpenAlex ID are matched by name, which can merge or split people.

Metadata source: OAI-PMH (metadata jurnal) https://ojs3.unpatti.ac.id/index.php/barekeng/oai, first harvested 5 Nov 2026, last seen 5 Nov 2026.
Original metadata (oai_dc)
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	<dc:title xml:lang="en-US">NUMERICAL ANALYSIS IN ARTERIAL STENOSIS AFFECTED BY ISCHEMIC HEART DISEASE USING FINITE VOLUME METHOD</dc:title>
	<dc:creator>Fatahillah, Arif</dc:creator>
	<dc:creator>Pratiwi, Alfiani Dyah</dc:creator>
	<dc:creator>Setiawani, Susi</dc:creator>
	<dc:creator>Kristiana, Arika Indah</dc:creator>
	<dc:creator>Adawiyah, Robiatul</dc:creator>
	<dc:description xml:lang="en-US">Atherosclerosis is the narrowing of blood vessels caused by the buildup of cholesterol plaque on the walls of the arteries. Excessive buildup of cholesterol plaque disrupts the circulatory system, thereby affecting the speed and pressure of blood flow. In the long term, atherosclerosis can cause ischemic heart disease. This study aims to analyze the influence of stenosis, initial velocity, and diameter on the velocity and pressure of blood flow in narrowed arteries that cause ischemic heart disease. In this research, a Navier Stoke mathematical equation model was built which was solved using the finite volume method with SIMPLE discretization (Semi Implicit Method for Pressure Linked Equations). Finite volume methods are used to analyze unstructured objects such as blood flow. SIMPLE discretization is implemented simply in two and three dimensions with a system of equations containing fluid motion. Matlab and Fluent are software used for process simulations, Matlab for visualizing graphs of numerical calculation results, and Fluent for visualizing blood flow. Based on the simulation results, it can be concluded that the smaller the diameter, the greater the stenosis, and the greater the initial velocity, the greater the blood flow velocity. On the contrary, the diameter gets smaller, the stenosis gets bigger, and the initial velocity gets bigger so that the pressure on the blood flow gets smaller. Blood flow simulation has the potential to contain ischemic heart disease if the maximum speed produced is greater than the maximum normal blood speed, namely 0.45 m/s.</dc:description>
	<dc:publisher xml:lang="en-US">PATTIMURA UNIVERSITY</dc:publisher>
	<dc:date>2024-03-01</dc:date>
	<dc:type>info:eu-repo/semantics/article</dc:type>
	<dc:type>info:eu-repo/semantics/publishedVersion</dc:type>
	<dc:format>application/pdf</dc:format>
	<dc:identifier>https://ojs3.unpatti.ac.id/index.php/barekeng/article/view/10138</dc:identifier>
	<dc:identifier>10.30598/barekengvol18iss1pp0179-0192</dc:identifier>
	<dc:source xml:lang="en-US">BAREKENG: Jurnal Ilmu Matematika dan Terapan; Vol 18 No 1 (2024): BAREKENG: Journal of Mathematics and Its Application; 0179-0192</dc:source>
	<dc:source>2615-3017</dc:source>
	<dc:source>1978-7227</dc:source>
	<dc:source>10.30598/barekengvol18iss1year2024</dc:source>
	<dc:language>eng</dc:language>
	<dc:relation>https://ojs3.unpatti.ac.id/index.php/barekeng/article/view/10138/7550</dc:relation>
	<dc:rights xml:lang="en-US">Copyright (c) 2024 Arif Fatahillah, Alfiani Dyah Pratiwi, Susi Setiawani, Arika Indah Kristiana, Robiatul Adawiyah</dc:rights>
	<dc:rights xml:lang="en-US">http://creativecommons.org/licenses/by-sa/4.0</dc:rights>
</oai_dc:dc>